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15th International Conference on Nephrology and Hypertension, will be organized around the theme “New advances for prevention/treatment in the field of Nephrology and Hypertension ”

Nephrologists Congress 2019 is comprised of keynote and speakers sessions on latest cutting edge research designed to offer comprehensive global discussions that address current issues in Nephrologists Congress 2019

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Renal nutrition is related to making sure that kidney patients consume right food to make dialysis efficient and improve health. Dialysis clinics have dietitians on workforce who assist patients plan meals. Popular tips are: ingesting greater high protein ingredients, and less excessive salt, high potassium, and high phosphorus foods. Patients also are counseled on secure fluid consumption tiers.

Metabolism is the chemical reactions involved in retaining the dwelling nation of the cells and the organism. Metabolism can be quite simply divided into classes:

Anabolism - the synthesis of all compounds needed through the cells

Catabolism - the breakdown of molecules to obtain energy

Metabolism is closely connected to vitamins and the provision of vitamins. Bioenergetics is a term which describes the biochemical or metabolic pathways by using which the cell in the long run obtains strength. Energy formation is one of the crucial components of metabolism

  • Track 1-1Renal Supplements
  • Track 1-2Nutrition Therapy
  • Track 1-3Renal function in living kidney donors
  • Track 1-4Platelet dysfunction
  • Track 1-5Fluid Intake

Hypertension (also called high blood pressure) happens when the force of your blood against your blood vessel walls increases enough to cause damage. People who have diabetes or long lasting kidney disease, blood pressure of 130/80 or more is thought about high. Some types of kidney disease may cause high blood pressure. More often it is high blood pressure that causes renal disease. High blood pressure can speed up the loss of kidney function in people with renal disease. Your doctor or nurse practitioner can tell how much renal damage you’ve had by measuring the amount of protein in your urine and estimating your total renal function from a simple blood test.

Nephrologists and hypertension experts work intimately with different authorities/specialists, including endocrinologists, cardiologists, medical caretakers, drug specialists, dietitians, educators and to convey high-quality, cost effective care to patients who experiences problems including the kidneys, complex blood pressure issues or uncontrolled hypertension.

As per a few investigations, hypertensive patients have a tendency to lose their renal capacity speedier than normotensive subjects. Furthermore, systolic as well as diastolic hypertension were accounted for to be major risk factors for end-stage renal failure (ESRF) in treated hypertensive patients.

  • Track 2-1Advances in nephrology
  • Track 2-2Strategies of Nephrology perspectives
  • Track 2-3Critical Nephrology
  • Track 2-4Secondary hypertension
  • Track 2-5Genetic factors and life style of high blood pressure
  • Track 2-6Hypertension and obesity

Clinical nephrology includes restorative treatment of renal diseases and conditions. This may involve working with different regions of the body that likewise are affected by the kidneys. Kidney issues may cause electrolyte imbalances in the circulatory system, bringing about intense side effects incorporating overabundance liquid in the tissues, mental confusion, irregular heartbeat. Clinical nephrology additionally can help patients who have hypertension, since hypertension can be damaging to the kidneys.

Patients who are in kidney failure as a result of damaged kidneys need dialysis to perform the functions the kidneys no longer can. A patient suffering with renal failure may need a kidney transplant. Clinical nephrology manages the process of dialysis, which is a system of removing waste blood from the body, cleaning the blood. Most people have two kidneys but can survive with only one as long as it's healthy.

  • Track 3-1Nephron Clinical Practice
  • Track 3-2Oncologic Nephrology
  • Track 3-3Urinaryincontinence/Enuresis
  • Track 3-4Stem Cell and Regenerative Nephrology
  • Track 3-5Critical Care Nephrology
  • Track 3-6The translation of research facility considers identified with kidney disease

Renal hypertension which is also called renovascular hypertension, is blood pressure caused by renal diseases. It can as a rule be controlled by some medications.
People with renal hypertension can be helped by angioplasty, stenting or surgery on the blood vessels of the kidney. A few people with renal hypertension can benefit from outside intervention by stenting, angioplasty, or surgery on the veins of the kidney.

Renovascular hypertension is hypertension because of narrowing of the arteries that convey blood to the kidneys. This condition is likewise called renal artery stenosis. The most well-known reason for renal vein stenosis is a blockage in the supply routes because of elevated cholesterol. This happens when a sticky, greasy substance rang plaque expands on the internal covering of the arteries, causing a condition known as atherosclerosis. At the point when the courses that convey blood to your kidneys wind up plainly restricted, less blood streams to the kidneys. The kidneys react as though your circulatory strain is low. Therefore, they discharge hormones that advise the body to clutch more salt and water. This causes your circulatory stream to rise.

  • Track 4-1Indications & symptoms of Renal Hypertension
  • Track 4-2Diagnosis & treatment
  • Track 4-3Pathogenesis of Renal Hypertension.
  • Track 4-4Risk factors for Renal Hypertension
  • Track 4-5Renal-artery angioplasty and stenting

Hypertension after renal transplantation is a strong predictor of patient and graft survival. In transplant recipients, hypertension is usually defined as blood pressure >140/90 mmHg or likewise if a patient is treated with antihypertensive drugs. Cardiovascular morbidity and mortality and shortened allograft survival are important consequences of inadequate control of hypertension. Hypertension in renal transplant recipients is common and ranges from 50% to 80% in adult recipients and from 47% to 82% in pediatric recipients. Donor and recipient factors, acute and chronic allograft injury, and immunosuppressive medications may each explain some of the pathophysiology of post-transplant hypertension. As observed in other patient cohorts, renal artery stenosis and adrenal causes of hypertension may be important contributing factors. Notably, BP treatment goals for renal transplant recipients remain an enigma because there are no adequate randomized controlled trials to support a benefit from targeting lower BP levels on graft and patient survival. The potential for drug-drug interactions and altered pharmacokinetics and pharmacodynamics of the different antihypertensive medications need to be carefully considered. To date, no specific antihypertensive medications have been shown to be more effective than others at improving either patient or graft survival. Identifying the underlying pathophysiology and subsequent individualization of treatment goals are important for improving long-term patient and graft outcomes in these patients.

  • Track 5-1Solid organ transplantation
  • Track 5-2Arterial hypertension
  • Track 5-3Organ transplant monitoring
  • Track 5-4Drug-drug interactions
  • Track 5-5Transplant coordinator

Hypertension  is most common among patients suffering with chronic kidney disease (CKD) and diabetes mellitus. Hypertension is predominant in most individuals with diabetic kidney disease (DKD). Failure to treat hypertension appropriately in this sub-group of patients result in an greater risk of cardiovascular morbidity and mortality, as well as a faster developing of kidney disease.

  • Track 6-1Risk factors for diabetes
  • Track 6-2Treatment and therapies.
  • Track 6-3Advanced treatments for diabetes
  • Track 6-4Destruction of the islet β-cell
  • Track 6-5Early management of obesity

There is much symptomatic similarity between intense kidney infection and intense coronary illness. Both may give shortness of breath and chest distress, and along these lines it isn't shocking that biomarkers of intense myocardial and  renal sickness frequently exist together in many specialists symptomatic work. The high occurrence of cardiovascular occasions in endless kidney illness warrants an exact assessment of hazard went for diminishing the actions of malady and its outcomes. The utilization of biomarkers to recognize patients at high hazard has been being used in the overall public for a very long while and has gotten blended responses in the medicinal group. A few specialists have turned out to be staunch supporters and clients while others question the utility of biomarkers and seldom measure them. In Chronic Kidney Disease patients various markers like those utilized as a part of the all-inclusive community and others more particular to the uremic populace have risen; however their utility for routine clinical application stays to be completely illustrated. The high frequency of cardiovascular illness (CVD) occasions and untimely mortality in patients with Kidney diseases with a sharp increment in chance as glomerular filtration rate (GFR) decays beneath 60 mL/min/1.72 m2 [2], offers a method of reasoning for better hazard stratification in this populace. A few conventional hazard factors and factors all the more firmly identified with loss of           (paleness, oxidative anxiety, aggravation, and bone mineral issue) add to the high rate of cardiovascular complexities found in patients with CKD. Regardless of whether biomarkers help enhance the recognizable proof of patients in danger of cardiovascular occasions has been at the center of broad research in the all inclusive community and in patients with CKD.

  • Track 7-1Hypertension and tachycardia
  • Track 7-2Glomerular filtration rate
  • Track 7-3Cardiovascular disease
  • Track 7-4Structural heart disease
  • Track 7-5Cardiac rehabilitation
  • Track 7-6Renal Cystic Diseases

Individuals from the Pediatric Nephrology and Hypertension division assess and treat children with intrinsic or acquired problems of the kidney and a wide range of hypertension. Pediatric nephrology offers expert examination, treatment and administration of a wide range of hypertensive and kidney issue in newborn children, young people, kids and. Masters incorporate talented and experienced pediatric nephrologists, pediatric medical caretakers, imaging authorities, and a group of numerous other multidisciplinary experts. Pediatric Nephrology and Hypertension experts gives discussion and care to children’s and infants with the full range of kidney ailments. This includes intrinsic anomalies of the urinary tract, acquired abnormalities, hereditary disorders, and hypertension.

  • Track 8-1Pediatric Renal Transplantation
  • Track 8-2Pediatric Kidney Dialysis
  • Track 8-3Kidney Care in children
  • Track 8-4Advances in Pediatric Kidney Operation
  • Track 8-5Pediatric Kidney Diseases

Nephrology Nursing is a deliberate effort to increase nursing knowledge by the discovery of latest facts through systematic enquiry. It includes improvement in patient care, reduced cost of kidney care provision, accountability and protection against litigation. Addition to the existing body of nursing knowledge enhancement of nursing as a profession.

Renal care nursing is the field of nursing with a focus on the most extreme consideration of the discriminatingly sick or unsteady chronic kidney patients. Contamination revulsion and nursing consideration is the control concerned with turning away nosocomial or health awareness related disease, a functional (as opposed to scholastic) sub-order of the study of disease transmission. Children’s who need escalated restorative consideration are regularly conceded into a unique region of the clinic called the Neonatal serious care and nursing consideration. The part of backing in discriminating nursing consideration: Critical consideration medical attendants work in a wide assortment of settings, filling numerous parts including bedside clinicians, attendant teachers, nursing analysts, clinical nursing authorities, nursing supervisors and medical attendant professionals. Measurements of Renal Care Nursing’s mission are to give attendants exact, current and applicable data and lodging to exceed expectations in discriminating consideration rehearse.

  • Track 9-1Primary Care Nephrology
  • Track 9-2Public Health and Community
  • Track 9-3Kidney Care Support Services
  • Track 9-4Nutrition and Chronic Kidney Disease
  • Track 9-5Nurse Educator
  • Track 9-6Clinical nurse specialist

Haemodialysis is a process of cleansing the blood of toxins, fluids and extra salt through a dialysis machine. It helps maintain proper chemical balance such as potassium, sodium and chloride and keeps the blood pressure under control. There are three styles of haemodialysis: Conventional haemodialysis, each day haemodialysis, and Nocturnal haemodialysis. The most recent dialysis machines available on the market are highly automatic and continuously display an array of protection-crucial parameters, which includes blood and dialysate drift prices; dialysis answer conductivity, temperature, and pH; and analysis of the dialysate for evidence of blood leakage or presence of air. The drug remedy followed  include supplements of vitamins, calcium, iron, phosphate binders, Antipruritics.

  • Track 10-1Conventional Haemodialysis
  • Track 10-2Daily Haemodialysis
  • Track 10-3Nocturnal Haemodialysis
  • Track 10-4Kidney Dialysis
  • Track 10-5Haemolytic Uraemic Syndrome
  • Track 10-6Vesico-ureteral Reflux

The management of hypertension in kidney diseases is for the most part in view of the administration of medications which can reduce proteinuria and to slowdowns the dynamic functional deterioration. Angiotensin-changing over chemical inhibitors (ACEI), which bring down both proteinuria and blood pressure, have developed as medications of choice in proteinuric patients with either typical renal function or mild to moderate  kidney failure. In non proteinuric nephropathies no controlled examinations exist showing the prevalence of ACEI over different medications. In these conditions calcium antagonists may likewise be utilized. The way to deal with patients with hypertension and renal illness ought to dependably take into consideration over the nature of the outcomes that are to be accomplished. If the aim is to lower proteinuria and slow down progression, at that point ACEI, conceivably connected with calcium adversaries, are the medications of choice.

  • Track 11-1Diabetes Management
  • Track 11-2Urogynecology
  • Track 11-3Glomerulonephritis
  • Track 11-4Lowering blood pressure
  • Track 11-5Maintenance therapy and reassessment

Kidney transplantation is the best treatment for patients with end-stage renal illness and is related with noteworthy enhancements in personal satisfaction and survival of patients with successful kidney joins. A kidney transplant is surgery to put a kidney from a giver into your body. Amid a transplant, the specialist puts the new kidney in your lower belly and associates the course and vein of the new kidney to your supply route and vein. Frequently, the new kidney will begin making urine when your blood begins moving through it. In any case, here and there it takes half a month to begin working. A contributor is a man who has passed away or a living individual, regularly a relative. A kidney from somebody who has dead is a deceased donor kidney. A kidney from a living individual is a living benefactor kidney. Specialists put most transplanted kidneys in the lower abdomen region, close to the crotch. The specialist interfaces the supply route and vein from the donar kidney to a conduit and a vein in your body so your blood moves through your new kidney. The specialist connects the ureter from the contributor kidney to your bladder, giving urine a chance to spill out of the new kidney to your bladder. The new kidney may begin working immediately or may take up to half a month to urine. In the event that the new kidney does not begin working immediately, you will require dialysis medicines to channel squanders and additional salt and liquid from your body until the point that it starts working. Unless your harmed kidneys cause contaminations or hypertension or are dangerous, they can stay in your body.

  • Track 12-1Advances in Kidney Transplantation
  • Track 12-2Transplantation Techniques
  • Track 12-3Pediatric Renal Transplantation
  • Track 12-4Pregnancy after Transplantation
  • Track 12-5Acute Renal Allograft Rejection
  • Track 12-6Stem Cell Transplantation

Changes in single nutrients such as reducing sodium have been the main focus on dietary interventions to prevent and treat hypertension. However, weight reduction, adopting the DASH eating pattern (which vegetables, emphasizes fruits, whole grains ,low-fat dairy foods, , poultry, fish and nuts and is low in saturated fat, total fat and cholesterol) and engaging in physical activity have potentially the same or greater effect on managing hypertension as sodium reduction.

The prevalence of hypertension is high and increasing worldwide. Drug therapy is effective, but for both "prehypertensive" and treated hypertensive patients, lifestyle changes are also important. Dietary modification is a key part of these changes, although skepticism about the role of diet in determining blood pressure has slowed implementation of the available guidelines. However, there is now a large body of evidence supporting a role for dietary salt, potassium, alcohol, and body mass in determining blood pressure.

  • Track 13-1Smoking cessation
  • Track 13-2Dietary potassium
  • Track 13-3Maintenance therapy and reassessment
  • Track 13-4Resources for promoting lifestyle management to patients
  • Track 13-5Salt sensitivity and resistance

Experimental Nephrology is devoted to the biology of the kidney (including developmental, cellular and molecular aspects) and to the cellular basis of renal diseases. Included are studies on the mechanisms of diseases and their responses to interventions. Its high-quality, peer-reviewed papers aim not only at documenting, but also at defining mechanisms of biological phenomena. Basic information on the cell biology and physiology of kidney cells is juxtaposed with the elucidation of molecular mechanisms of renal pathophysiology and immunological function which characterize the spectrum of renal diseases. Managed by a dedicated editorial board comprising both basic biological scientists and clinical nephrologists, Experimental Nephrology represents a unique voice in renal cell biology.

  • Track 14-1Current Pathological Perspectives
  • Track 14-2Nephron
  • Track 14-3Clinical research Nephrologists
  • Track 14-4Fetal medicine

A case report is a method for conveying something new that has been learnt from clinical practice. It could be around a surprising or beforehand obscure condition, an uncommon presentation or difficulty of a known illness, or even another way to deal with dealing with a typical condition. A case report gives the point by point report of indications, signs, conclusion, treatment, and follow-up of an individual patient. Case reports may contain a statistic profile of the patient and assume significant part in the field of restorative clinical research and prove based pharmaceutical. Besides, case reports can serve as an early cautioning signal for the antagonistic impacts of new solutions, or the presentations of new and rising ailments.

  • Track 15-1Case reports dealing with the use of novel technologies
  • Track 15-2Case reports in oncology, diabetes and cardiovascular diseases
  • Track 15-3Dermatology, gastroenterology, Nephrology, pulmonology and urology case reports.
  • Track 15-4Case reports in neurology, ophthalmology, obstetrics and gynecology

Glomerulonephritis is a group of diseases that damage the part of the kidney that filters the blood (called glomeruli). And in other terms you it is also called as nephritis and nephrotic syndrome. Glomerulonephritis take place on its own or as part of alternative disease, such as lupus or diabetes. Severe or prolonged inflammation related with glomerulonephritis can also damage the kidneys in humans. The symptoms commonly seen in glomerulonephritis patients are pink or cola-colored urine (hematuria), foamy urine due to excess protein (proteinuria),  and  the Fluid retention (edema) with  the swelling of face, hands, feet and abdomen.

  • Track 16-1Nephritis
  • Track 16-2Nephrotic syndrome
  • Track 16-3Acute post-streptococcal glomerulonephritis
  • Track 16-4Primary glomerulonephritis
  • Track 16-5Estimated glomerular filtration rate (GFR)
  • Track 16-6Glomerular Filtration Rate
  • Track 16-7Severe glomerular injury

Lupus nephritis is aggravation of the kidney that is caused by foundational lupus erythematous (SLE). Likewise called lupus, SLE is an immune system malady. With lupus, the body's invulnerable framework focuses on its own body tissues. Lupus nephritis happens when lupus includes the kidneys. Lupus nephritis is an intense issue. Its side effects, however, are not generally emotional. Not all urinary or kidney issues in individuals with lupus are because of lupus nephritis. Individuals with lupus may likewise be inclined to urinary tract diseases. Indeed, even with treatment, loss of kidney work once in a while advances. In the event that both kidneys come up short, individuals with lupus nephritis may require dialysis. Dialysis includes sifting the blood through a machine to expel squander items from the body.

  • Track 17-1Systemic lupus erythematous
  • Track 17-2Lupus flare
  • Track 17-3Lupus erythematosus
  • Track 17-4Protein in the urine

Kidney failure, also called end-stage renal disease (ESRD), is the last stage of chronic kidney disease. When your kidneys fail, it means they have stopped working well enough for you to survive without dialysis or a kidney transplant. End-stage kidney or renal disease (ESRD) is the final stage of chronic kidney disease in which the kidneys no longer function well enough to meet the needs of daily life. People with diabetes or hypertension have the highest risk of developing ESRD. The treatments for ESRD are dialysis or kidney transplant. End-stage kidney disease is also called end-stage renal disease (ESRD). The kidneys of people with ESRD function below 10 percent of their normal ability, which may mean they’re barely functioning or not functioning at all. ESRD is the fifth stage of the progression of chronic kidney disease, which is measured by your glomerular filtration rate (GFR)

Certain people are at higher risk of developing ESRD, such as people who have:



Relatives with ESRD

  • Track 18-1Chronic Kidney Disease Diagnosis
  • Track 18-2Infection
  • Track 18-3Dementia
  • Track 18-4Anemia

Acute kidney injury (AKI) is a sudden episode of kidney failure or kidney damage that happens within a few hours or a few days. AKI causes a build-up of waste products in your blood and makes it hard for your kidneys to keep the right balance of fluid in your body. AKI can also affect other organs such as the brain, heart, and lungs. Acute kidney injury is common in patients who are in the hospital, in intensive care units, and especially in older adults.

Some disease and conditions can damage your kidneys and lead to Acute Kidney Injury. Some examples include:

  • An allergic reaction to certain types of drugs (called “interstitial nephritis”)
  • A type of cancer called “multiple myeloma
  • A group of diseases (called “scleroderma”) that affect the connective tissue that supports your internal organs
  • A rare condition that causes inflammation and scarring to your blood vessels, making them stiff, weak, and narrow (called “vasculitis”)
  • Conditions that cause inflammation or damage to the kidney tubules, to the small blood vessels in the kidneys, or to the filtering units in the kidneys (such as “tubular necrosis,” “glomerulonephritis, “vasculitis” or “thrombotic microangiopathy”).
  •  A type of severe, life-threatening infection called “sepsis”
  • Track 19-1Prerenal Acute renal Failure
  • Track 19-2Postrenal Acute renal Failure
  • Track 19-3Intrinsic renal failure
  • Track 19-4Acute Kidney Injury– Experimental Models
  • Track 19-5Tubular Diseases
  • Track 19-6Renal filtration function